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Alleviation of Autophagic Deficits and Neuroinflammation by Histamine H3 Receptor Antagonist E159 Ameliorates Autism-Related Behaviors in BTBR Mice
Shilu Deepa Thomas
, Petrilla Jayaprakash
, Nurfirzana Z.H.J. Marwan
, Ezzatul A.B.A. Aziz
, Kamil Kuder
, Dorota Łażewska
, Katarzyna Kieć-Kononowicz
,
Bassem Sadek
Department of Pharmacology and Therapeutics
Research output
:
Contribution to journal
›
Article
›
peer-review
5
Citations (Scopus)
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Dive into the research topics of 'Alleviation of Autophagic Deficits and Neuroinflammation by Histamine H3 Receptor Antagonist E159 Ameliorates Autism-Related Behaviors in BTBR Mice'. Together they form a unique fingerprint.
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Pharmacology, Toxicology and Pharmaceutical Science
Nervous System Inflammation
100%
Histamine H3 Receptor Antagonist
100%
Autism
100%
Compulsion
75%
Alzheimer's Disease
25%
Cytokine
25%
Histamine H3 Receptor
25%
Brain Disease
25%
Beclin 1
25%
Histamine H3 Receptor Agonist
25%
Autophagy Related Protein
25%
Systemic Treatment
25%
Immune Dysregulation
25%
Biochemistry, Genetics and Molecular Biology
H3 Receptor Antagonist
100%
Autophagy
66%
Histamine H3 Receptor
66%
Proinflammatory Cytokine
33%
Receptor Agonists
33%
Synaptic Transmission
33%
Social Interaction
33%
Immunity
33%
Histaminergic
33%
Neuroscience
Autism
100%
Autophagy Related Protein
25%
Brain Disease
25%
Keyphrases
Behavioral Dysregulation
25%
LC3A
25%
Behavior Deficits
25%