Endoglin mutants retained in the endoplasmic reticulum exacerbate loss of function in hereditary hemorrhagic telangiectasia type 1 (HHT1) by exerting dominant negative effects on the wild type allele

Nesrin Gariballa, Sally Badawi, Bassam R. Ali

Research output: Contribution to journal › Article › peer-review

5 Citations (Scopus)

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Biochemistry, Genetics and Molecular Biology

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