TY - GEN
T1 - Health risk of inhaled (nano)-particles
AU - Hoet, P. H.M.
AU - Nemmar, A.
AU - Nemery, B.
PY - 2004
Y1 - 2004
N2 - The effect of inhaled particles on haemostasis and cardiovascular endpoints such as thrombosis was studied. Two complementary hypotheses were proposed to explain cardiovascular dysfunction after inhalation of ultrafine particles. It has been shown that within an hour after the deposition of diesel exhaust particle (DEP) in the lungs, both a marked pulmonary inflammation and enhanced femoral venous and arterial thrombosis are apparent. The results indicate that nano-sized particles can, after the passage into the circulation, also play a role, and therefore rapid interact with cardiovascular functions such as thrombogenesis.
AB - The effect of inhaled particles on haemostasis and cardiovascular endpoints such as thrombosis was studied. Two complementary hypotheses were proposed to explain cardiovascular dysfunction after inhalation of ultrafine particles. It has been shown that within an hour after the deposition of diesel exhaust particle (DEP) in the lungs, both a marked pulmonary inflammation and enhanced femoral venous and arterial thrombosis are apparent. The results indicate that nano-sized particles can, after the passage into the circulation, also play a role, and therefore rapid interact with cardiovascular functions such as thrombogenesis.
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M3 - Conference contribution
AN - SCOPUS:13844276046
SN - 1877040193
SN - 9781877040191
T3 - Transactions - 7th World Biomaterials Congress
SP - 751
BT - Transactions - 7th World Biomaterials Congress
T2 - Transactions - 7th World Biomaterials Congress
Y2 - 17 May 2004 through 21 May 2004
ER -
