On the interaction of β-Amyloid peptides and α7-Nicotinic acetylcholine receptors in Alzheimer's disease

Pharmacology, Toxicology and Pharmaceutical Science

• Amyloid 100%
• Receptor 75%
• Oligomer 50%
• Neurofibrillary Tangle 25%
• Amyloid Plaque 25%
• Monomer 25%
• Desensitization 25%
• Tau Protein 25%
• Neuroprotective Agent 25%

Keyphrases

• Amyloid-β Peptide 100%
• Neurochemicals 10%
• Amyloid Plaques 10%
• Neurofibrillary Tangles 10%
• Colocalization 10%
• Neurodegenerative 10%
• Tau Phosphorylation 10%
• Homomeric 10%
• Nanomolar Concentrations 10%
• Long-term Potentiation 10%
• Neuroprotective 10%
• Vascular Smooth muscle Cells 10%
• Dose Dependence 10%
• Rapid Desensitization 10%
• Astrocytes 10%
• Hyperphosphorylated tau Protein 10%
• Induced Toxicity 10%
• Hippocampus-dependent Memory 10%
• Receptor Activation 10%

Neuroscience

• Nicotinic Acetylcholine Receptor 100%
• Long-Term Potentiation 25%
• Astrocyte 25%
• Vascular Smooth Muscle 25%
• Tau Protein 25%
• Neuroprotective Agent 25%