Waterpipe Smoke Inhalation Induces Lung Injury and Aortic Endothelial Dysfunction in Mice

Pharmacology, Toxicology and Pharmaceutical Science

• Endothelial Dysfunction 100%
• Lung Injury 100%
• Glutathione 66%
• Tumor Necrosis Factor 66%
• Interleukin 1 66%
• Nuclear Factor 66%
• Sirtuin 1 66%
• Superoxide Dismutase 33%
• Interleukin 6 33%
• Inflammation 33%
• Cytokine 33%
• E-Selectin 33%
• Vascular Cell Adhesion Molecule 1 33%
• Intercellular Adhesion Molecule 1 33%
• Nitric Oxide 33%
• Caspase 3 33%
• Atherosclerosis 33%
• Comet Assay 33%
• Galectin 3 33%
• Lipid 33%
• Cell Adhesion Proteins 33%

Keyphrases

• Lung Injury 100%
• Waterpipe Smoking 100%
• Endothelial Dysfunction 100%
• Smoke Inhalation 100%
• Inhalation 50%
• Apoptosis 20%
• Glutathione 20%
• Tumor Necrosis Factor-α 20%
• Interleukin-1β 20%
• Nuclear factor-κB 20%
• Silent Information Regulator 1 (SIRT1) 20%
• Lipid Peroxidation 10%
• Superoxide Dismutase 10%
• Neutrophils 10%
• Macrophages 10%
• Interleukin-6 10%
• Nitric Oxide 10%
• Inflammatory Cytokines 10%
• Oxidative Stress Markers 10%
• Galectin-3 (Gal-3) 10%
• Oxidative Inflammation 10%
• P-selectin 10%
• DNA Damage 10%
• Intercellular Adhesion molecule-1 (ICAM-1) 10%
• Vascular Cell Adhesion molecule-1 (VCAM-1) 10%
• E-selectin 10%
• Adhesion Molecules 10%
• Bronchoalveolar Lavage Fluid 10%
• Cleaved caspase-3 10%
• Comet Assay 10%
• Aortic Tissue 10%
• Tobacco Smoking Exposure 10%
• Endothelial Inflammation 10%
• Exposed Groups 10%